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Inhibiting the production of the antioxidant molecule glutathione shows potential for treating a common form of deadly esophageal cancer
Alternatively, gene ontology and pathway analysis in tissues indicated increased protein intensities related to important tissue functionalities
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The small molecule APR-246 (PRIMA-1Met) can reactive mutant p53 in tumor cells and trigger cell death by apoptosis
L-PAM depleted GSH, but GSH rapidly recovered in a L-PAM-resistant MM cell line unless also treated with BSO
