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The relationship between oxidative stress and inflammation is an essential physiological response of the body to various disease states, such as pathogen invasion, tissue damage/injury, and irritants

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Neuron-specific PHD2 inactivation reduces brain injury and functional impairment after acute stroke through HIF-dependent mechanisms The infarct lesion size was significantly reduced in neuron-specific Phd2 deficient ( nPhd2 / ) mice, while brain swelling did not differ markedly between nPhd2 / and Phd2 f/f mice subjected to acute ischemic stroke (Figure 1C)

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