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However, such therapy must not interfere with standard treatments, including antimicrobial therapy and hemodynamic stabilization with vasoactive drugs
Together, these findings demonstrate that, in contrast with acute exposure, chronic disruption of iron homeostasis with consequent GSH depletion remodels cellular redox homeostasis over time, inducing a state we term chronoferroptosis: a persistent ferroptotic adaptation characterized by coordinated alterations in iron-handling and antioxidant defense proteins that may represent early vulnerability to neurodegenerative pathology
We report the dependence of glioma cells on exogenous cysteine/cystine, despite this amino acid being nonessential
