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ROS As the therapeutic impact of high-dose Vit-C is strongly dependent on the formation of ROS, it is essential to define this term

The CCK-8 results showed that cell viability decreased in a concentration- and time-dependent manner following emodin treatment at both 24 and 48 h, compared with the control group ( Figure 1A ), suggesting that emodin may inhibit the proliferation of HCCLM3 cells

Beyond mitochondrial and enzymatic ROS sources, bioactive metal dyshomeostasis may contribute to A/Tau-mediated radical generation and OS (Beal, 2013

They affect how many satellite cells are available, how efficiently nutrients move into the fiber, and how strongly the intracellular pathways respond afterward

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