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A series of recent studies implicated sirtuins as potent negative regulators of fibroblast activation and myofibroblast differentiation, and sirtuin dysfunction has been linked to the pathogenesis of SSc and other fibro-inflammatory conditions 28,29,38,39,40,41,42
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Through allosteric antagonism, Isatuximab effectively inhibits both CD38 hydrolase and cyclase activity [81]
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