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During catagen induction, proliferation in matrix cells ceases and subsequently programed cell death is initiated within the matrix in close proximity to the DP 8

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A randomized, placebo-controlled trial of coenzyme Q10 and remacemide in Huntington's disease

conducted a study which identified phenotypic changes in primary cultures of coronary arterial myocytes derived from CD38 KO mice, revealing an increase in vimentin expression, suggesting de-differentiated of vascular smooth muscle, and an increase in cell proliferation [32]