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Intracranial (superior sagittal sinus) hypertension, portal and caval hypertension, aortal hypotension, progressing venous and arterial thrombosis peripherally and centrally, and ECG disturbances [14,15,16,20,22,23,24,25] were commonly counteracted

The reason is bioavailability and dosing consistency

[163, 165] have reported that off-target toxicity in peripheral organs remained minimal, although they emphasised that excessively high doses or positively charged CQDs may still provoke ROS formation, mitochondrial dysfunction, or the activation of inflammatory pathways [163, 165]

Boyles AL, Yetley EA, Thayer KA, Coates PM

The primary antidepressant mechanism involves promoting serotonin accumulation in the cerebral cortex