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Correspondingly, KEAP1 undergoes conformational changes and disconnects from Nrf2 under oxidative stress conditions, promoting Nrf2 translocated to nucleus and combined with antioxidant response element (ARE) to regulating the transcriptional expression of detoxification genes, thereby preventing oxidative damage, mitochondrial damage and ATP depletion [44]
IGF-1 LR3 is hypothesized to mitigate these risks by supporting neuronal survival and improved cardiac output
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Rosmarinic acid
Vitamin C - Antioxidant Power: Helps protect cells from oxidative damage
