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Acknowledgements We express our gratitude to the Vice-Chancellor for Research, Isfahan University of Medical Sciences for financial support through the scientific code 398979 and grant number 1989096

Toxic necrosis of the epidermis

doi: 10.1007/s00264-018-4211-5 54

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As superoxide is subject to efficient dismutation by cellular superoxide dismutases, it is possible that NFT might directly interact with Keap1 to induce the observed release of Nrf2 and its escape from proteasomal degradation