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Abstract Cognitive decline frequently occurs with increasing age, but mechanisms contributing to age-associated cognitive decline (ACD) are not well understood and solutions are lacking

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doi:10.1016/S0892-0362(01)00119-2

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Here, by preventing the interaction of HIF1- with E3 ubiquitin ligases, both VEGF and CD31 expression was enhanced in ECs, coinciding with increased cell proliferation and migration collectively accelerating wound healing in diabetic mice 64