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In this review, we provide an mtROS-centered framework to bridge this gap by integrating anatomical and histopathological changes with mitochondrial dysfunction, redox dysregulation, and catabolic signaling in rotator cuff injury-induced supraspinatus atrophy

Similar content being viewed by others Introduction Pancreatic cancer(PC) is one of the most lethal malignancies with a 5-year survival rate less than 9% [1]

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protection against free radicals and toxins

The peptide mechanisms of action are not gender-specific, and the safety profile appears comparable across genders
