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The primary underlying pathomechanism is most likely a decreased hepatic GHR expression related to insulin deficiency, resulting in GH-resistance and impaired hepatic IGF-1 synthesis, which stimulates pituitary GH-secretion by a feedback mechanism [188, 189]

Older individuals with greater baseline synaptic loss may show more pronounced responses to a given dose than younger individuals with intact synaptic density, but this reflects neurobiological substrate, not chronological age itself

11 , e37576 (2022)

Targeting and crossing the blood-brain barrier with extracellular vesicles

Liu J, Clough SJ, Hutchinson AJ et al (2016) MT1 and MT2 melatonin receptors: a therapeutic perspective