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An alteration in the phosphorylation pattern, such as an increase in tyrosine phosphorylation, which results from pathological conditions, such as inflammation or elevated ROS, can alter the proteinprotein interactions of occludin with ZO-1, ZO-2, and ZO-3, and thereby alter the membrane integrity 75

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This infection leads to inflammatory demyelination in brain and spinal cord, with a mixture of cluster of differentiation (CD)4 + and CD8 + T-cells, B-cells, microglia, and macrophages contributing to the demyelination [14]

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