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After heavy metal was removed, the memory of expression developed, and heavy metal-transporting P-type ATPase genes (HMAs) were activated due to heavy metal-associated stress ( Figure 2 illustrates the epigenetic regulation of stress-responsive genes across abiotic stresses

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Consistently, S34P mutation of the rat amylin backbone peptide significantly increased peptide affinity for amylin receptor 1 ECD binding by 3.6-fold (Figure 2E and Table 2)

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