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Interestingly, we found that a surface-exposed cysteine residue (Cys 63 ) located in the thioredoxin domain of Ec CnoX, away from the SXXC motif, is involved in the formation of mixed-disulfide complexes with substrate proteins under HOCl stress (more than 130 proteins were identified), thereby protecting them from irreversible damage which could otherwise block reactivation (Goemans et al., 2018b)

First in vivo evidence of an NMDA receptor deficit in medication-free schizophrenic patients

Wachter, J

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