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C., Garty G., et al., Biofluid Metabolomics and Lipidomics of Mice Exposed to External Very HighDose Rate Radiation, Metabolites 12, no

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In this review we will analyze the role of GSH in the intestine, the molecular mechanisms by which GSH depleting drugs inhibit the intestinal Ca 2+ absorption and the prevention or restoration of these effects by drugs that act through normalization of intestinal GSH content

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VE, as a fat-soluble antioxidant, can significantly lower levels of ROS and lipid peroxidation products in the brain, thereby preserving the structural integrity of the BBB [124]