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Notably, S100 protein expression was persistently negative in this case, suggesting tumor dedifferentiation, epigenetic dysregulation, and malignant transformation
[30] They allow endogenously released acetylcholine more time to interact with its respective receptor before being inactivated by acetylcholinesterase in the synaptic cleft (the space between nerve and muscle)
1996 PMID: 8831529 Dorr RT, et al
doi:10.1093/bioinformatics/btr190
The glucose effect is dose-related and reversible with discontinuation in most cases
