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In conclusion, the in vivo work on Gstp / mice fully demonstrates that GSTP acts as a negative regulator in sepsis pathophysiology, and recombinant GSTP protein treatment to mice may significantly improve survival in mouse CLP sepsis model
Traumatic brain injury causes chronic cortical inflammation and neuronal dysfunction mediated by microglia
In conclusion, FeS clusters are essential for electron transfer and CO 2 reduction in biology
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Thus, Trx1 may also serve as a potential diagnostic marker and be useful in prognostic assessments
