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The objectives of this review are to (i) propose biologically plausible explanation(s) of how carnitine metabolism could contribute to the development and/or maintenance of an AUD and (ii) identify limitations and gaps in the literature available surrounding carnitine metabolism defects and AUDs as a basis for future study

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In hypothyroidism-associated MAFLD, aberrant ECM remodeling alters not only matrix composition but also its biomechanical properties, particularly stiffness

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