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Although the exact role and mechanism of autophagy in the metabolic coupling between TME and AML still require comprehensive and in-depth research, the existing evidence suggests that targeting autophagy to disrupt the metabolic coupling between TME and AML may represent a promising strategy for circumventing the protective effects of stromal cells in AML

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[70], Gazia et al

Budesonide GI-Directed Corticosteroid Considered when symptoms suggest intestinal inflammation alongside mast cell involvement

Developmental exposure to methylmercury elicits early cell death in the cerebral cortex and long-term memory deficits in the rat