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Consequently, despite persistently high circulating levels of FGF21, its metabolic and protective actions are largely blunted

Therefore, even though little is known about the control of SAMe levels in the brain, the neuronal concentration of SAMe can be further investigated as a key factor in the molecular and behavioral responses to the stressors [71]

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It has been demonstrated that enhanced glutamine metabolism results in the accumulation of toxic ammonia levels, which in turn damages lysosomes and mitochondria, leading to T-cell depletion