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However, this robust inhibition did not affect the concentration of M0 C3 or total C3 acylcarnitine, suggesting that the contribution of valine catabolism to the pool of C3 acylcarnitine is limited by the low expression level of BCAT in plated human hepatocytes and that other substrates make key contributions to these distal pools of intermediates

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[1] It has been suggested that the peptide may have the potential to influence diverse physiological processes like feeding patterns, central nervous system operations, blood pressure, and more

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