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Recent studies have linked TXN system damage with hyperoxic injury and abnormal lung development, noting that the TXN system not only has a simple ROS detoxification function but also acts as a REDOX sensor when oxygen concentration changes, assisting newborns to complete the oxygen concentration conversion process from intrauterine relative hypoxia to hyperoxia exposure (67)

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These findings suggest that ellagitannins may modulate disease development through the gut microbiota-bile acid axis