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Nicergoline has been shown to be an effective drug for preventing neuronal vulnerability due to experimentally induced nerve growth factor deprivation and improved the function of cholinergic and catecholaminergic neurotransmitters in rats in vivo [64]

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This was further supported by studies showing that the FERM and SH2 domains of JAK1 are essential for association to oncostatin M receptor (OSMR), and that this binding is also required for upregulating surface expression of the OSMR (66, 67)