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Chronic Myelogenous Leukemia: ASH 2007 Update

3.7 Coagulation and other pathways activated in CHL We observed a significant upregulation of proteins in CHL compared to polyp, specifically those involved in blood coagulation and fibrin clot formation ( p adjusted = 7.1E-08, GO_BP), formation of fibrin clot (clotting cascade) ( p adjusted = 4.5E-07, REAC), common pathway of fibrin clot formation ( p adjusted = 2.9E-05, REAC), and blood clotting cascade ( p adjusted = 0.0001, WP) (Supplementary Table S6

reduced glutathione (GSH) is the active form that is able to repair oxidative damage, and oxidized glutathione (GSSG) is the inactive form that can be recycled back into GSH

In muscle injury model studies, muscle injury (such as trauma, exerciseinduced injury, or diseaserelated damage) triggers a series of pathophysiological processes including myofiber rupture, inflammatory responses, and muscle stem cell activation