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The protein actin is necessary for these cells to migrate to the wound site [1]

iPLA2-mediated lipid detoxification controls p53-driven ferroptosis independent of GPX4
EGCG has protective properties and activates the Shh signaling pathway in PC12 cells damaged by CORT

The location of the SMHT1 gene (17p11.2) resides within the 3.7 Mb region of chromosome 17 that is deleted in Smith-Magenis syndrome, SMS, a disorder associated with behavioral problems, psychomotor impairment, growth delay, speech delay, brachycephaly, midface hypoplasia, and a hoarse voice

Furthermore, SLC1A5 overexpression in female mice induces polycystic ovary syndrome-like phenotypes, including hyperandrogenism and abnormal follicle development
