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The biosynthesis of glutathione (GSH) and the normal function of GPX4 are central to the control of ferroptosis, and the inhibition of GPX4 can increase cellular ferroptosis sensitivity [6, 7]

Schilling, D

In addition, enteral or parenteral cystine, methionine, N-acetyl-cysteine, and L-2-oxothiazolidine-4-carboxylate are effective precursors of cysteine for tissue GSH synthesis [84]

Consequently, mGSH arises from the cytosol GSH into the mitochondrial matrix by a carrier-mediated transport located in the MIM that overcomes the unfavorable entry against an electrochemical gradient (26, 45, 78, 96) (Fig

Association between blood lead level and subsequent Alzheimers disease mortality