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This review interprets HTY-related mechanisms through direct chemical actions such as radical scavenging and metal chelation, indirect cellular associations indicated by alterations in downstream markers (e.g., Nrf2, NF-B, AMPK-related changes), and translational relevance, evidenced when effects become evident at physiologically attainable exposures and/or in human studies

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Developmental increase in vesicular glutamate content does not cause saturation of AMPA receptors at the calyx of Held synapse

Redox sensing and signaling associated with reactive oxygen in chloroplasts, peroxisomes and mitochondria

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