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Cell-based therapies, particularly those that leverage the capacity of stem cells for decidualization or epithelial differentiation, offer a potential solution to improve endometrial function

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Moreover, the p53 4KR98 mutant (R98/R117/R161/R162) failed to repress SLC7A11 transcription or induce ferroptosis [68], suggesting a critical role of p53 acetylation in SLC7A11-related ferroptosis

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