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Atg7 -deficient microglia fail to sustain the DAM signature (downregulating Clec7a , Trem2 , and Tyrobp ) and instead adopt a senescence-associated microglial (SAM) phenotype

Also, the ER membrane disequilibrium activates UPR ER in yeast, mammalian cells, and mice [86,91]

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Post conditioning demonstrates a viable surgical option, especially in the early stages of reperfusion when testis damage is frequent 42