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Uricase-based gout treatments are generally limited due in part to the accumulation of H2O2 in an arthrosis microenvironment
This activation reduces the inhibitory binding between Caveolin-1 and eNOS, allowing greater eNOS activation and nitric oxide productionwith laboratory studies showing BPC-157 at 1 g/ml increases NO production 1.35-fold compared to controls
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