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They hypothesized that the expression of ferroptosis-protective genes was an adaptive response to IR therapy, given that the expression levels rose subsequently to IR-induced induction of pro-ferroptotic genes such as ACSL4 [35]
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Cysteine S -nitrosylation protects protein-tyrosine phosphatase 1B against oxidation-induced permanent inactivation
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[PubMed] [Google Scholar] 172.Shekhter A.B., Soloveva A.I., Spevak S.E., Titov M.I
