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Exposure of astrocytes to various -amyloid peptides, and -amyloid 2535 in particular, increased Cx43 expression and appears to redistribute connexins from gap junction assemblies to hemichannels 486,488,489 , the latter, in turn, mediating excessive release of glutamate and ATP resulting in excitotoxicity 489,490,491

Glutathione binding to the Bcl-2 homology-3 domain groove: a molecular basis for BCL-2 antioxidant function at mitochondria

GHK-Cu appears to regulate copper in the brain, preventing the aggregation of amyloid beta (A), a key factor in Alzheimers disease

doi: 10.1007/BF00445792 79

Proteins and peptides that have free amino group(s) (in the side chain) will also react and give colour with ninhydrin