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doi: 10.1021/acs.analchem.9b05630
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doi: 10.1038/s41590-021-00996-0 38 DrijversJMGillisJEMuijlwijkTNguyenTHGaudianoEFHarrisISet al
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Using the pressure dependent model, we can therefore hypothesize that high levels of ApoC-II on the TRL surface would increase the surface pressure preventing LPL binding, thereby attenuating lipolysis, when ApoC-II is overexpressed
