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A significant change in the bench press third set lifting volume at week 6 was observed in the LCR group (146 kg, 95% CI 21.1, 272) but not in the PLA group (65.2 kg, 95% CI -65.7, 196)

It could be shown that apoC1 can induce apoptosis in cultured VSMCs via a mechanism involving neutral sphingomyelinase-ceramide pathway [129], and that apoC1 co-localized with markers of apoptosis (ceramide, caspase-3) in neo-intimal dissections in rabbit models of atherosclerosis [130]

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