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However, the convergence of axonal loss in both NCS classifications also suggests that a unified, targeted treatment approach that addresses complement-mediated nerve damage driven by autoantibodies against either myelin or axons can be effective for all patients with GBS
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10.1155/2018/9076485 177 OTooleP
Research findings have demonstrated that GSH acts as a methylglyoxal detoxifier that confers salt stress tolerance (Singla-Pareek et al
