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GLY I converts a highly cytotoxic metabolite methylglyoxal (MG) into S-lactoylglutathione (SLG) with the help of reduced glutathione (GSH) 3 .The SLG is further hydrolyzed into D-lactate by GLY II and GSH is recycled back into the system (Fig

5fC: Formyl-cytosine

The amino-acid sequence of dDAT in the primary binding site has high similarity to hNET and hDAT (Supplementary Fig

Therefore, thyroid dysfunction, including abnormalities in thyroid hormone synthesis and the emergence of autoimmune thyroid conditions such as Graves disease and Hashimotos thyroiditis, has been linked to Se deficiency