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ACO2 deficiency increases vulnerability to Parkinsons disease via dysregulating mitochondrial function and histone acetylation-mediated transcription of autophagy genes

doi:10.1016/j.ygcen.2012.04.001

Correction to: molecular and pharmacological modulators of the tumor immune contexture revealed by deconvolution of RNA-seq data
On the other hand, during energy deficit conditions, AMPK is activated, which inhibits mTORC1, thereby activating ULK1 [90]

The pigment cells are both a target and a promoter of complement and these actions must be highly regulated to support ocular immune privilege
