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These results emphasize the elaborate relationship between histone methylation and cancer development, highlighting the potential of these enzymes as therapeutic targets for cancer treatment (Table 1)

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35 Homozygous (biallelic) ABCB4 transporter mutations and compound heterozygous mutations lead to higher rates of progression to cirrhosis, as seen in PFIC3

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Significantly, although methionine is identified as an important factor of nutritional epigenomics for maintaining T cell function, the tumor-derived or exogenous SAM metabolite also reprograms chromatin accessibilities of CD8 + T cells, leading to T cell dysfunction (50)