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The pathogenesis of atopic dermatitis involves a complex interplay between skin barrier dysfunction and type 2 inflammation, among other factors 2,6-8 Dysregulated IL-4 and IL-13 signaling may exacerbate epidermal barrier dysfunction, which contributes to the clinical manifestations of disease The unique role of IL-4 in type 2 inflammation IL-4 is an orchestrator of Th2 response, creating a positive feedback loop that leads to increased secretion of IL-4, IL-13, and IL-31 9,a,b a Type 2 cytokines (such as IL-4, IL-13, and IL-31) may also be produced by other immune cells, such as ILC2