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One possible explanation is that hyperoxidized PRXs in the SRX mutant could either signal as damage associated molecular patterns (DAMPs) themselves or alter post-translation modifications of other proteins that in turn signal as DAMPs, to induce beneficial response pathways 53

GPx4 plays an essential role in neurodegeneration by inhibiting ferroptosis, which is iron-dependent cell death and recently implicated in numerous diseases [4,5]

Particularly for rarer cancers, such as thyroid cancer, it is essential to use large, population-based studies such as these to identify potential risk factors. Source: Oxford University 15.09.2020

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