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( b ) In vitro mechanistic validation demonstrates the superiority of the dual-gene strategy
Likewise, the overexpression of SOD is children with Down syndrome leads to a reduction of GSH and an increase in oxidative stress [13]
It is caused by increased activity of cyclin-dependent kinase inhibitors (p16 and p21), leading to resistance to apoptosis and metabolic changes, including accumulation of senescence-associated -galactosidase (SA--gal) [73]
In contrast to the oxidation of reduced roGFP2, AtGrxS15 did not catalyse the reduction of oxidized roGFP2 in the presence of GSH (Supplementary Fig
Radiother Oncol
