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The following is a summary of documented tissue systems from peer-reviewed literature: This cross-tissue activity profile is directly attributable to the fact that its primary mechanisms VEGFR2-driven angiogenesis and NO system modulation are not tissue-specific

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This may be another potential mechanism for action of ED medicines, as most ED cases are associated with oxidative stress [55]

3603) and GFP fluorescence was recorded on a Biotek Synergy 2 plate reader

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