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However, the obtained data suggested a more complex inhibitory process than that of squalamine, involving the displacement of -Syn monomers from the lipid vesicles and the interaction with aggregation intermediates [300]
Several studies have suggested the paracrine activation of c-MET/HGF signaling in HNSCC [81]
This includes the following: Notable cognitive and physical improvements mark the subacute phase as the brain continues to heal and adapt
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