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While the wholesale induction or inhibition of autophagy may have deleterious side effects, the still-theoretical ability to activate or inhibit the autophagic degradation of a specific cancer-related target could be considerably safer since only some of autophagy's many physiological roles would be impacted

For instance, healthy liver function requires a number of things like glutathione to detox naturally and cope with the stress and inflammation from toxins

ALS-associated mutation FUS-R521C causes DNA damage and RNA splicing defects

EDIT: Your post intrigued me and I was a bit concerned about the assumption that normal ferritin levels meant that haemochromotosis is not present