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Mechanically, atractylenolide-III promotes the upregulation of the autophagy markers Beclin1, LC3, and ULK1 and autophagic degradation of Keap1, by which activating the Nrf2/ARE pathway and inhibiting inflammation and oxidative stress [174]

Mutations at positions 47, 51, 74, 78, 82, 87, 188, 263, 328, 330, and 353 have been frequently associated with improved hydroxylation of aromatic substrates, as evidenced by multiple references to each position

Moreover, AIs are used off-label to reduce circulating estradiol levels, thereby inhibiting the negative feedback of AAS use, limiting excessive testosterone release

Z., Wu J., et al

doi: 10.3390/molecules26092540 39