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The finding that a long-acting GIP receptor agonist (LAGIPRA) increases insulin sensitivity by promoting glucose capture in WAT confirms this
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10.1046/j.1432-1327.1999.00780.x 5 BirsoyK.WangT.ChenW
GHK modulated an activity of both metalloproteinases and their inhibitors (TIMP-1 and TIMP-2), acting as a main regulator of wound healing and skin remodeling processes [7, 8]
Thus, dysfunctional activation of the UPR in melanocytes may cause more immunogenesis, which induces an attack on melanocytes in vitiligo
