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Agents such as mitochondrial stabilizers, anti-inflammatory drugs or compounds that enhance energy metabolism can help counteract oxysterol-driven oxidative stress and apoptotic signaling

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The NF-B pathway functions as a central pro-survival and pro-inflammatory transcription factor that promotes tumor progression by suppressing tumor suppressors such as phosphatase and tensin homolog (PTEN), thereby indirectly enhancing PI3K/AKT signaling and supporting cancer cell viability