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The clone in red was named LaccID

However, when antioxidative systems become saturated, RBCs accumulate oxidative lesions, provoking biochemical alterations and advancing pathological developments ( 2 O 2 can diffuse out of erythrocytes and provoke inflammatory activation in the lungs microvascular endothelium ( 2 O 2 and hydroxyl radicals inflict damage on erythrocyte membrane proteins and lipids, resulting in reorganization of the cytoskeleton, loss of membrane stability and deformability, and eventually eryptosis (Sudnitsyna et al., 2020)

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