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Increased serotonin at postsynaptic 5-HT1 A receptors in the dorsal horn of spinal cord inhibits the release of nociceptive neurotransmitters from sensory neurons, resulting in analgesia [9, 17]
The results from this study highlight the critical role that glutathione homeostasis has on modulating exercise induced oxidative stress, and conversely, the effect of oxidative stress at rest on exercise induced changes in glutathione redox status[17]
Spence et al
The idea that all risk must be fully documented before you're allowed to choose is not how medicine actually works
Vitamin C helps "recharge" glutathione once it has been used by the body, allowing it to stay active longer
